NEW YORK (GenomeWeb) – Researchers at Stanford College and the Massachusetts Institute of Know-how used a mixture of tumor barcoding, CRISPR-Cas9-mediated genome modifying, and ultra-deep barcode sequencing to interrogate pairwise combos of mutations in tumor suppressor genes in autochthonous mouse fashions of human lung adenocarcinoma in an effort to decide the practical affect of those alterations.

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